By Nancy Appleton, PhD Contributed by Nancy Appleton, PhD
Author of the book Lick The Sugar Habit
In addition to throwing off the body's homeostasis, excess sugar may result in a number of other significant consequences. The following is a listing of some of sugar's metabolic consequences from a variety of medical journals and other scientific publications.
1. Sugar can suppress your immune system and impair your defenses against infectious disease.1,2
2. Sugar upsets the mineral relationships in your body: causes chromium and copper deficiencies and interferes with absorption of calcium and magnesium. 3,4,5,6
3. Sugar can cause can cause a rapid rise of adrenaline, hyperactivity, anxiety, difficulty concentrating, and crankiness in children.7,8
4. Sugar can produce a significant rise in total cholesterol, triglycerides and bad cholesterol and a decrease in good cholesterol.9,10,11,12
5. Sugar causes a loss of tissue elasticity and function.13
6. Sugar feeds cancer cells and has been connected with the development of cancer of the breast, ovaries, prostate, rectum, pancreas, biliary tract, lung, gallbladder and stomach.14,15,16,17,18,19,20
7. Sugar can increase fasting levels of glucose and can cause reactive hypoglycemia.21,22
8. Sugar can weaken eyesight.23
9. Sugar can cause many problems with the gastrointestinal tract including: an acidic digestive tract, indigestion, malabsorption in patients with functional bowel disease, increased risk of Crohn's disease, and ulcerative colitis.24,25,26,27,28
10. Sugar can cause premature aging.29
11. Sugar can lead to alcoholism.30
12. Sugar can cause your saliva to become acidic, tooth decay, and periodontal disease.31,32,33
13. Sugar contributes to obesity.34
14. Sugar can cause autoimmune diseases such as: arthritis, asthma, multiple sclerosis.35,36,37
15. Sugar greatly assists the uncontrolled growth of Candida Albicans (yeast infections).38
16. Sugar can cause gallstones.39
17. Sugar can cause appendicitis.40
18. Sugar can cause hemorrhoids.41
19. Sugar can cause varicose veins.42
20. Sugar can elevate glucose and insulin responses in oral contraceptive users.43
21. Sugar can contribute to osteoporosis.44
22. Sugar can cause a decrease in your insulin sensitivity thereby causing an abnormally high insulin levels and eventually diabetes.45,46,47
23. Sugar can lower your Vitamin E levels.48
24. Sugar can increase your systolic blood pressure.49
25. Sugar can cause drowsiness and decreased activity in children.50
26. High sugar intake increases advanced glycation end products (AGEs)(Sugar molecules attaching to and thereby damaging proteins in the body).51
27. Sugar can interfere with your absorption of protein.52
28. Sugar causes food allergies.53
29. Sugar can cause toxemia during pregnancy.54
30. Sugar can contribute to eczema in children.55
31. Sugar can cause atherosclerosis and cardiovascular disease.56,57
32. Sugar can impair the structure of your DNA.58
33. Sugar can change the structure of protein and cause a permanent alteration of the way the proteins act in your body.59,60
34. Sugar can make your skin age by changing the structure of collagen.61
35. Sugar can cause cataracts and nearsightedness.62,63
36. Sugar can cause emphysema.64
37. High sugar intake can impair the physiological homeostasis of many systems in your body.65
38. Sugar lowers the ability of enzymes to function.66
39. Sugar intake is higher in people with Parkinson's disease.67
40. Sugar can increase the size of your liver by making your liver cells divide and it can increase the amount of liver fat.68,69
41. Sugar can increase kidney size and produce pathological changes in the kidney such as the formation of kidney stones.70,71
42. Sugar can damage your pancreas.72
43. Sugar can increase your body's fluid retention.73
44. Sugar is enemy #1 of your bowel movement.74
45. Sugar can compromise the lining of your capillaries.75
46. Sugar can make your tendons more brittle.76
47. Sugar can cause headaches, including migraines.77
48. Sugar can reduce the learning capacity, adversely affect school children's grades and cause learning disorders.78,79
49. Sugar can cause an increase in delta, alpha, and theta brain waves which can alter your mind's ability to think clearly.80
50. Sugar can cause depression.81
51. Sugar can increase your risk of gout.82
52. Sugar can increase your risk of Alzheimer's disease.83
53. Sugar can cause hormonal imbalances such as: increasing estrogen in men, exacerbating PMS, and decreasing growth hormone.84,85,86,87
54. Sugar can lead to dizziness.88
55. Diets high in sugar will increase free radicals and oxidative stress.89
56. High sucrose diets of subjects with peripheral vascular disease significantly increases platelet adhesion.90
57. High sugar consumption of pregnant adolescents can lead to substantial decrease in gestation duration and is associated with a twofold increased risk for delivering a small-for-gestational-age (SGA) infant.91,92
58. Sugar is an addictive substance.93
59. Sugar can be intoxicating, similar to alcohol.94
60. Sugar given to premature babies can affect the amount of carbon dioxide they produce.95
61. Decrease in sugar intake can increase emotional stability.96
62. Your body changes sugar into 2 to 5 times more fat in the bloodstream than it does starch.97
63. The rapid absorption of sugar promotes excessive food intake in obese subjects.98
64. Sugar can worsen the symptoms of children with attention deficit hyperactivity disorder (ADHD).99
65. Sugar adversely affects urinary electrolyte composition.100
66. Sugar can slow down the ability of your adrenal glands to function.101
67. Sugar has the potential of inducing abnormal metabolic processes in a normal healthy individual and to promote chronic degenerative diseases.102
68. I.V.s (intravenous feedings) of sugar water can cut off oxygen to your brain.103
69. Sugar increases your risk of polio.104
70. High sugar intake can cause epileptic seizures.105
71. Sugar causes high blood pressure in obese people.106
72. In intensive care units: Limiting sugar saves lives.107
73. Sugar may induce cell death.108
74. In juvenile rehabilitation camps, when children were put on a low sugar diet, there was a 44 percent drop in antisocial behavior.109
75. Sugar dehydrates newborns.110
76. Sugar can cause gum disease.111

References
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2. Ringsdorf, W., Cheraskin, E. and Ramsay R. Sucrose, Neutrophilic Phagocytosis and Resistance to Disease, Dental Survey. 1976;52(12):46_48.
3. Couzy, F., et al. "Nutritional Implications of the Interaction Minerals," Progressive Food and Nutrition Science 17;1933:65-87
4. Kozlovsky, A., et al. Effects of Diets High in Simple Sugars on Urinary Chromium Losses. Metabolism. June 1986;35:515_518.
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37. Erlander, S. The Cause and Cure of Multiple Sclerosis, The Disease to End Disease." Mar 3, 1979;1(3):59_63.
38. Crook, W. J. The Yeast Connection. (TN:Professional Books, 1984).
39. Heaton, K. The Sweet Road to Gallstones. British Medical Journal. Apr 14, 1984; 288:00:00 1103_1104. Misciagna, G., et al. American Journal of Clinical Nutrition. 1999;69:120-126.
40. Cleave, T. The Saccharine Disease. (New Canaan, CT: Keats Publishing, 1974).
41. Ibid.
42. Cleave, T. and Campbell, G. (Bristol, England:Diabetes, Coronary Thrombosis and the Saccharine Disease: John Wright and Sons, 1960).
43. Behall, K. Influ ence of Estrogen Content of Oral Contraceptives and Consumption of Sucrose on Blood Parameters. Disease Abstracts International. 1982;431437.
44. Tjäderhane, L. and Larmas, M. A High Sucrose Diet Decreases the Mechanical Strength of Bones in Growing Rats. Journal of Nutrition. 1998:128:1807_1810.
45. Beck, Nielsen H., Pedersen O., and Schwartz S. Effects of Diet on the Cellular Insulin Binding and the Insulin Sensitivity in Young Healthy Subjects. Diabetes. 1978;15:289_296 .
46. Sucrose Induces Diabetes in Cat. Federal Protocol. 1974;6(97). diabetes
47. Reiser, S., et al. Effects of Sugars on Indices on Glucose Tolerance in Humans. American Journal of Clinical Nutrition. 1986;43:151-159.
48. Journal of Clinical Endocrinology and Metabolism. Aug 2000
49. Hodges, R., and Rebello, T. Carbohydrates and Blood Pressure. Annals of Internal Medicine. 1983:98:838_841.
50. Behar, D., et al. Sugar Challenge Testing with Children Considered Behaviorally Sugar Reactive. Nutritional Behavior. 1984;1:277_288.
51. Furth, A. and Harding, J. Why Sugar Is Bad For You. New Scientist. Sep 23, 1989;44.
52. Simmons, J. Is The Sand of Time Sugar? LONGEVITY. June 1990:00:00 49_53.
53. Appleton, N. New York: LICK THE SUGAR HABIT. Avery Penguin Putnam:1988. allergies
54. Cleave, T. The Saccharine Disease: (New Canaan Ct: Keats Publishing, Inc., 1974).131.
55. Ibid. 132
56. Pamplona, R., et al. Mechanisms of Glycation in Atherogenesis. Medical Hypotheses . 1990:00:00 174_181.
57. Vaccaro O., Ruth, K. J. and Stamler J. Relationship of Postload Plasma Glucose to Mortality with 19 yr Follow up. Diabetes Care. Oct 15,1992;10:328_334. Tominaga, M., et al, Impaired Glucose Tolerance Is a Risk Factor for Cardiovascular Disease, but Not Fasting Glucose. Diabetes Care. 1999:2(6):920-924.
58. Lee, A. T. and Cerami, A. Modifications of Proteins and Nucleic Acids by Reducing Sugars: Possible Role in Aging. Handbook of the Biology of Aging. (New York: Academic Press, 1990.).
59. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology 1990:45(4):105_110.
60. Cerami, A., Vlassara, H., and Brownlee, M. Glucose and Aging. Scientific American. May 1987:00:00 90
61. Dyer, D. G., et al. Accumulation of Maillard Reaction Products in Skin Collagen in Diabetes and Aging. Journal of Clinical Investigation. 1993:93(6):421_22.
62. Veromann, S.et al."Dietary Sugar and Salt Represent Real Risk Factors for Cataract Development." Ophthalmologica. 2003 Jul-Aug;217(4):302-307.
63. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI
64. Monnier, V. M. Nonenzymatic Glycosylation, the Maillard Reaction and the Aging Process. Journal of Gerontology. 1990:45(4):105_110.
65. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
66. Appleton, Nancy. New York; Lick the Sugar Habit. Avery Penguin Putnam, 1988 enzymes
67. Hellenbrand, W. Diet and Parkinson's Disease. A Possible Role for the Past Intake of Specific Nutrients. Results from a Self-administered Food-frequency Questionnaire in a Case-control Study. Neurology. Sep 1996;47(3):644-650.
68. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38.
69. Ibid.
70. Yudkin, J., Kang, S. and Bruckdorfer, K. Effects of High Dietary Sugar. British Journal of Medicine. Nov 22, 1980;1396.
71. Blacklock, N. J., Sucrose and Idiopathic Renal Stone. Nutrition and Health. 1987;5(1-2):9- Curhan, G., et al. Beverage Use and Risk for Kidney Stones in Women. Annals of Internal Medicine. 1998:28:534-340.
72. Goulart, F. S. Are You Sugar Smart? American Fitness. March_April 1991:00:00 34_38. Milwakuee, WI,:
73. Ibid. fluid retention
74. Ibid. bowel movement
75. Ibid. compromise the lining of the capillaries
76. Nash, J. Health Contenders. Essence. Jan 1992; 23:00 79_81.
77. Grand, E. Food Allergies and Migraine.Lancet. 1979:1:955_959.
78. Schauss, A. Diet, Crime and Delinquency. (Berkley Ca; Parker House, 1981.)
79. Molteni, R, et al. A High-fat, Refined Sugar Diet Reduces Hippocampal Brain-derived Neurotrophic Factor, Neuronal Plasticity, and Learning. NeuroScience. 2002;112(4):803-814.
80. Christensen, L. The Role of Caffeine and Sugar in Depression. Nutrition Report. Mar 1991;9(3):17-24.
81. Ibid,44
82. Yudkin, J. Sweet and Dangerous.(New York:Bantam Books,1974) 129
83. Frey, J. Is There Sugar in the Alzheimer's Disease? Annales De Biologie Clinique. 2001; 59 (3):253-257.
84. Yudkin, J. Metabolic Changes Induced by Sugar in Relation to Coronary Heart Disease and Diabetes. Nutrition and Health. 1987;5(1-2):5-8.
85. Yudkin, J and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988:32(2):53-55.
86. The Edell Health Letter. Sept 1991;7:1.
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88. Journal of Advanced Medicine. 1994;7(1):51-58.
89. Ceriello, A. Oxidative Stress and Glycemic Regulation. Metabolism. Feb 2000;49(2 Suppl 1):27-29.
90. Postgraduate Medicine.Sept 1969:45:602-07.
91. Lenders, C. M. Gestational Age and Infant Size at Birth Are Associated with Dietary Intake among Pregnant Adolescents. Journal of Nutrition. Jun 1997;1113- 1117
92. Ibid.
93. Sugar, White Flour Withdrawal Produces Chemical Response. The Addiction Letter. Jul 1992:04:00 Colantuoni, C., et al. Evidence That Intermittent, Excessive Sugar Intake Causes Endogenous Opioid Dependence. Obes Res. Jun 2002 ;10(6):478-488. Annual Meeting of the American Psychological Society, Toronto, June 17, 2001 www.mercola.com/2001/jun/30/sugar.htm
94. Ibid.
95. Sunehag, A. L., et al. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition Diabetes. 1999 ;48 7991_800.
96. Christensen L., et al. Impact of A Dietary Change on Emotional Distress. Journal of Abnormal Psychology.1985;94(4):565_79.
97. Nutrition Health Review. Fall 85 changes sugar into fat faster than fat
98. Ludwig, D. S., et al. High Glycemic Index Foods, Overeating and Obesity. Pediatrics. March 1999;103(3):26-32.
99. Pediatrics Research. 1995;38(4):539-542. Berdonces, J. L. Attention Deficit and Infantile Hyperactivity. Rev Enferm. Jan 2001;4(1)11-4
100. Blacklock, N. J. Sucrose and Idiopathic Renal Stone. Nutrition Health. 1987;5(1 & 2):9-
101. Lechin, F., et al. Effects of an Oral Glucose Load on Plasma Neurotransmitters in Humans. Neurophychobiology. 1992;26(1-2):4-11.
102. Fields, M. Journal of the American College of Nutrition. Aug 1998;17(4):317_321.
103. Arieff, A. I. Veterans Administration Medical Center in San Francisco. San Jose Mercury; June 12/86. IVs of sugar water can cut off oxygen to the brain.
104. Sandler, Benjamin P. Diet Prevents Polio. Milwakuee, WI,:The Lee Foundation for for Nutritional Research, 1951
105. Murphy, Patricia. The Role of Sugar in Epileptic Seizures. Townsend Letter for Doctors and Patients. May, 2001 Murphy Is Editor of Epilepsy Wellness Newsletter, 1462 West 5th Ave., Eugene, Oregon 97402
106. Stern, N. & Tuck, M. Pathogenesis of Hypertension in Diabetes Mellitus. Diabetes Mellitus, a Fundamental and Clinical Test. 2nd Edition, (PhiladelphiA; A:Lippincott Williams & Wilkins, 2000)943-957.
107. Christansen, D. Critical Care: Sugar Limit Saves Lives. Science News. June 30, 2001; 159:404.
108. Donnini, D. et al. Glucose May Induce Cell Death through a Free Radical-mediated Mechanism.Biochem Biohhys Res Commun. Feb 15, 1996:219(2):412-417.
109. Schoenthaler, S. The Los Angeles Probation Department Diet-Behavior Program: Am Empirical Analysis of Six Institutional Settings. Int J Biosocial Res 5(2):88-89.
110. Gluconeogenesis in Very Low Birth Weight Infants Receiving Total Parenteral Nutrition. Diabetes. 1999 Apr;48(4):791-800.
111. Glinsmann, W., et al. Evaluation of Health Aspects of Sugar Contained in Carbohydrate Sweeteners." FDA Report of Sugars Task Force -1986 39 123 Yudkin, J. and Eisa, O. Dietary Sucrose and Oestradiol Concentration in Young Men. Annals of Nutrition and Metabolism. 1988;32(2):53-5.
CNN reports what some of us have known for a long time now — by making changes such as adding exercise and improving their diets, many type 2 diabetics can drop their glucose or sugar numbers back to the normal range, reversing their condition.
Diabetes is caused when your body isn’t making enough insulin, or it can’t properly use the insulin it is producing. As a result of this, too much sugar stays in the blood, which can cause serious problems.
According to CNN: “People who carry excess weight, especially in their midsection, are more likely to develop type 2 diabetes because the fat in their tissues causes an imbalance of insulin in the body. The condition is called insulin resistance. If they can eliminate that fat by exercising and limiting carbohydrates and alcohol, then many can drop their glucose levels. And for some, they can drop them back into the normal range.”
I’ve been educating readers on how to reverse type 2 diabetes for years now, and conventional media has finally caught up with this rare report. The conventional drug treatment for this disease has done nothing but exacerbate the problem, and a large portion of the standard diabetes recommendations are downright disastrous. Together, drugs and improper dietary advice has caused diabetics to die prematurely.
According to the National Diabetes Fact Sheet, 2011 from the US Centers for Disease Control and Prevention (CDC), type 1 and 2 diabetes now affects 25.8 million people, or 8.3 percent of the U.S. population. That number skyrockets in older age groups: nearly 27 percent of American seniors had diabetes in 2010. If you add in pre-diabetics (insulin resistance), the number jumps to more than 1 in 4 Americans across age groups!
While type 1 diabetes is an autoimmune disease that shuts down your body’s insulin production, type 2 diabetes is directly caused by an unhealthy lifestyle. Type 1 diabetics do need to inject insulin several times a day to stay alive, but type 2 diabetics do NOT need drugs. In fact, taking insulin if you’re a type 2 diabetic is one of the WORST things you can do. Any physician recommending insulin regularly for type 2 diabetes suffers from profound ignorance of insulin physiology.
Your Diet Will Make You or Break You, Especially if You’re Diabetic
This is one of the very few conventional media reports I’ve seen that actually gets it right, and doesn’t advocate some sort of flawed dietary advice. The subject of their story, Jonathan Lugg, did exactly what needed to be done. He cut out most of the sugar and carbs from his diet (including alcohol, which also equates to sugar), and increased his protein intake, along with regular exercise.
Why does this work?
Because this combination corrects and restores your body’s insulin/leptin signaling.
For the last 50 years or so, Americans have followed the dietary recommendations of a high complex carbohydrate, low saturated fat diet — the exact opposite of what actually works! High complex carbohydrates include legumes, potatoes, corn, rice and grain products. Aside from legumes, you actually want to AVOID all the rest to prevent insulin resistance.
“Conventional wisdom” also states that table sugar is okay for diabetics, as long as you readjust your medications to compensate appropriately. And using toxic artificial sweeteners like aspartame in lieu of sugar also gets the green light, despite the evidence showing it rapidly stimulates the release of insulin and leptin (which diabetics need to avoid), and actually lead to greater weight gain than sugar…
Unfortunately, industry lobbying and various conflicts of interest have a lot to do with why diabetics have been given such flawed advice for so long.
- Leptin is a hormone produced in your fat cells. One of leptin’s primary roles is regulating your appetite and body weight. It tells your brain when to eat, how much to eat, and most importantly, when to stop eating. And leptin tells your brain what to do with the energy it has. Leptin is largely responsible for the accuracy of insulin signaling and whether or not you become insulin resistant.
- Insulin — Sugars and grains (complex carbs) raise your blood sugar. When this happens, insulin is released to direct the extra energy into storage. A small amount is stored as a starch called glycogen, but the majority is stored as your main energy supply — fat. Insulin’s major role is not to lower your blood sugar, but rather to store the extra energy for future times of need. Insulin's effect of lowering your blood sugar is merely a u201Cside effectu201D of this energy storage process.
If you consume loads of sugars and grains, your blood sugar spikes will lead to increased insulin, which leads to increased fat storage. The extra fat then produces more leptin. The problem arises when your leptin levels become chronically elevated. At this point, you become leptin resistant — your body can no longer “hear” the hormonal signals telling your brain you’re full and should stop eating.
As your fat stores increase, your weight goes up, and insulin resistance sets in. Now your body has become “deaf” to the signals from both hormones (leptin and insulin), and disease follows; one of which is diabetes.
Why Conventional Diabetes Treatment is All Wrong
The mechanism described above explains why diabetes treatments concentrating merely on lowering blood sugar can actually worsen, rather than remedy the actual problem of metabolic miscommunication. This is also why taking insulin is one of the WORST things you can do for type 2 diabetes, since it will actually worsen your insulin and leptin resistance over time.Diabetes is a well-known risk factor for a number of other serious diseases, including heart attack and stroke, for which additional drug therapy is typically prescribed. However, four different studies published last year concluded these drug therapies are useless at best and dangerous at worst.
They found that:
- Using antihypertensives to lower systolic blood pressure does nothing to lower a diabetic’s risk of heart complications
- Diabetics get no health benefit from adding a drug to raise HDL “good” cholesterol levels if they're already taking a statin to lower their LDL cholesterol levels
- There were no heart benefits associated with two different drugs given to lower high blood sugar levels
Top 10 Guidelines for Preventing and Reversing Type 2 Diabetes
Following these simple guidelines can help you do at least three things that are essential for successfully treating diabetes: recover your insulin/leptin sensitivity; help normalize your weight; and naturally normalize your blood pressure:- Severely limit or eliminate grains and sugar from your diet, especially fructose which is far more detrimental than any other type of sugar. This is extremely important! Drinking just one sweetened drink a day can raise your diabetes risk by 25 percent compared to drinking one sugary drink per month, so you really need to evaluate your diet and look for hidden sources of sugar.
This also means avoiding most processed foods of all kinds as they are loaded with fructose. You may even need to avoid fruits until your blood sugar is under control.
- Following my nutritional type diet will help you do this without much fuss. I now offer the entire online nutritional typing test for free, which makes it even easier.
It's important to realize that while nearly all type 2 diabetics need
to swap out their grains for other foods, some will benefit from using
protein for the substitution, while others will benefit from using more
vegetable-only carbohydrates. Therefore, along with reducing grains and
sugars, determining your nutritional type will give you some insight
into what foods you should use to replace them.
- Exercise is an absolutely essential factor, without which you’re highly unlikely to get this devastating disease under control. It is clearly one of the most potent ways to lower your insulin and leptin resistance. Make sure to incorporate high-intensity sprint-type exercises. These types of exercises boost fat loss, promote muscle building, and make your body produce human growth hormone (HGH) naturally. Typically, you’ll need large amounts of exercise until you get your blood sugar levels under control. You may need up to an hour or two a day. Naturally, you’ll want to gradually work your way up to that amount, based on your current level of fitness.
- Avoid trans fats as they will actually worsen insulin resistance.
- Consume saturated fats,
such as grass-fed organic meat, raw dairy products, avocados, and
coconut oil. These saturated fats provide a concentrated source of
energy along with the building blocks for cell membranes and a variety
of hormones and hormone-like substances. When you eat healthy fats as
part of your meal, they slow down absorption so that you can go longer
without feeling hungry. In addition, they act as carriers for important
fat-soluble vitamins A, D, E and K.
There are more than a dozen different types of saturated fat,
but you predominantly consume only three: stearic acid, palmitic acid
and lauric acid. It's already been well established that stearic acid
(found in cocoa and animal fat) has no effect on your cholesterol levels
at all, and actually gets converted in your liver into the
monounsaturated fat called oleic acid.
The other two, palmitic and lauric acid, do raise total cholesterol. However, since they raise u201Cgoodu201D cholesterol as much or more than u201Cbadu201D cholesterol, you're still actually lowering your risk of heart disease.
- Get plenty of omega-3 fats from a high-quality, animal-based source.
- Monitor your fasting insulin level. This is every bit as important as your fasting blood sugar. You’ll want your fasting insulin level to be between 2 to 4. The higher your level, the worse your insulin receptor sensitivity is. The recommendations mentioned above are the key steps you need to achieve this reduction.
- Get enough high-quality sleep every night.
- Optimize your vitamin D levels. Maintaining your vitamin D levels around 60–80 ng/ml can significantly help control your blood sugar.
In addition, recent studies have revealed that getting enough vitamin D can also have a powerful effect on normalizing your blood pressure, and reduces your risk of heart disease.
Having optimal vitamin D levels can also prevent type 1 diabetes in your children if you are pregnant. It’s also vital for infants to receive the appropriate amounts of vitamin D in their early years for the same reasons. Ideally, you’ll want to do this by exposing a large amount of your skin to appropriate amounts of sunshine (or a safe tanning bed) on a regular basis, year-round. Your body can safely create up to 20,000 units of vitamin D a day this way. If neither of these options are available, you can use an oral vitamin D3 supplement. Just remember to get your levels tested regularly by a proficient lab to make sure you’re staying within the therapeutic range.
- Address any underlying emotional issues and/or stress. Non-invasive tools like the Emotional Freedom Technique/Meridian Tapping Technique can be extremely helpful and effective.